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1.
为了对边坡的合理设计提供严密的参考依据,采用原位孔内剪切试验测试了某路基边坡填土的抗剪强度,对不同工况下的边坡支护效果开展数值模拟分析.结果表明,该路基边坡填土压实质量欠佳,呈现轻微的应变硬化特征,其内摩擦角为24°,黏聚力为12 kPa.采用桩基托梁挡墙支护后,边坡最大横向位移可以降低76.5%~84.5%,且随着桩长的增加,边坡变形逐渐减小,但减小幅度呈下降趋势;边坡全局应变水平相比支护前也有所减小,局部最大剪应变明显降低,滑弧特征被削弱.因此,可采用原位孔内剪切试验较为准确地获取边坡填土的强度参数,使得边坡支护设计更合理. 相似文献
2.
Existing Side-Channel Attacks(SCAs) have several limitations and, rather than to be real attack methods,can only be considered to be security evaluation methods. Their limitations are mainly related to the sampling conditions, such as the trigger signal embedded in the source code of the encryption device, and the acquisition device that serves as the encryption-device controller. Apart from it being very difficult for an attacker to add a trigger into the original design before making an attack or to control the encryption device, there is a big gap in the capacity of existing SCAs to pose real threats to cipher devices. In this paper, we propose a new method, the sliding window SCA(SW-SCA), which can be applied in scenarios in which the acquisition device is independent of the encryption device and for which the encryption source code requires no trigger signal or modification. First,we describe the main issues in existing SCAs, then we theoretically analyze the effectiveness and complexity of our proposed SW-SCA —a method that can incorporate a sliding-window mechanism into almost all of the existing non-profiled SCAs. The experimental results for both simulated and physical traces verify the effectiveness of the SW-SCA and the appropriateness of its theoretical complexity. 相似文献
3.
To investigate the attack and defense strategies in complex networks, the authors propose a two-player zero-sum static game model with complete information which considers attack and defense strategies simultaneously. The authors assume that both the attacker and defender have two typical strategies: Targeted strategy and random strategy. The authors explore the Nash equilibriums of the attacker-defender game and demonstrate that when the attacker's attack resources are not so significantly abundant as the defender's resources, there exists a pure-strategy Nash equilibrium in both model networks and real-world networks, in which the defender protects the hub targets with large degrees preferentially, while the attacker prefers selecting the targets randomly. When the attack resources are much higher than defense resources, both the attacker and the defender adopt the targeted strategy in equilibriums. This paper provides a new theoretical framework for the study of attack and defense strategies in complex networks. 相似文献
4.
根据重塑纯黏土和砂-黏土混合物试样的室内固结试验结果,反演了渗透系数,探讨了纯黏土和砂-黏土混合物的渗透特性差异.试验结果表明:常用于评价纯黏土渗透性状的归一化指标e/eL(孔隙比与液限状态孔隙比的比值)能拓展运用于砂-黏土混合物中,但e/eL与渗透系数之间呈现非线性关系,这显著不同于纯黏土的线性关系,其本质原因在于混合物在压缩过程中形成了砂骨架.骨架形成前,纯黏土和砂-黏土混合物的e/eL与渗透系数的2条关系曲线吻合良好,而在骨架形成后,两者的关系曲线存在明显的差异;引入砂-黏土混合物四相体系,从黏土孔隙比和砂孔隙比的角度阐述了砂骨架的形成机制,且对两者渗透性状的差异性进行了详细的机理解释;最后给出了一种可以预测纯黏土和砂-黏土混合物渗透系数的经验公式. 相似文献
5.
目的探讨尼莫地平对SAH大鼠脑功能改善作用,并分析相关机制.方法采用枕大池二次注血法建立大鼠SAH模型,通过神经行为学评分,经颅多普勒超声检测基底动脉的平均血流速度,HE染色法观察脑形态学改变,脑含水量检测分析尼莫地平对SAH的治疗机制.结果与假手术组相比,SAH组大鼠出现明显的神经运动功能障碍体征,其神经行为学评分明显降低;与SAH组相比,尼莫地平组大鼠神经运动功能障碍体征明显减轻,神经行为评分明显高于SAH组.颅超声多普勒检测结果表明:SAH组大脑基底动脉血流速度明显加快,而经过尼莫地平治疗后,其血流速度明显降低;HE染色发现:SAH组大鼠脑中可见明显的出血灶,神经元数目减少,神经元及神经胶质出现肿胀,尼莫地平能够明显减轻SAH后的脑损伤;大鼠SAH时,脑水含量明显增加,尼莫地平既能减轻血管源性脑水肿症状,又减轻细胞毒性脑水肿症状.结论尼莫地平能改善神经运动功能障碍,其机制与降低基底动脉血流速度、减轻神经细胞、胶质细胞肿胀及减轻脑水肿有关. 相似文献
6.
7.
The flotation of hemimorphite using the S(Ⅱ)–Pb(Ⅱ)–xanthate process,which includes sulfidization with sodium sulfide,activation by lead cations,and subsequent flotation with xanthate,was investigated.The flotation results indicated that hemimorphite floats when the S(Ⅱ)–Pb(Ⅱ)–xanthate process is used; a maximum recovery of approximately 90% was obtained.Zeta-potential,contact-angle,scanning electron microscopy–energy-dispersive spectrometry(SEM–EDS),and diffuse-reflectance infrared Fourier transform spectroscopy(DRIFTS) measurements were used to characterize the activation products on the hemimorphite surface and their subsequent interaction with sodium butyl xanthate(SBX).The results showed that a Zn S coating formed on the hemimorphite surface after the sample was conditioned in an Na2 S solution.However,the formation of a Zn S coating on the hemimorphite surface did not improve hemimorphite flotation.With the subsequent addition of lead cations,Pb S species formed on the mineral surface.The formation of the Pb S species on the surface of hemimorphite significantly increased the adsorption capacity of SBX,forming lead xanthate(referred to as chemical adsorption) and leading to a substantial improvement in hemimorphite flotation.Our results indicate that the addition of lead cations is a critical step in the successful flotation of hemimorphite using the sulfidization–lead ion activation–xanthate process. 相似文献
8.
耕作过程中保持耕深稳定是提高耕作质量的重要措施之一.以小型耕耘机的后悬挂旋耕部件为研究对象,提出了一种基于积分分离式PID控制的耕深自动测量和调节的系统,采用双倾角传感器检测耕深,消除了田块自身与绝对水平面倾角的误差影响,通过在误差信号的处理中使用积分分离式PID控制算法,利用Simulink中的SimHydraulic模块对耕深控制系统进行动态仿真研究,与未加入PID控制算法时的仿真结果进行比较,发现几乎消除了系统的耕深超调现象.同时,将响应时间由4.0s左右缩短到1.4s左右,使耕深控制系统的动态响应达到较好的效果.田间试验结果表明,当耕深设定为10cm和16cm时,实测耕深稳定性变异系数分别为6.05%和3.54%,均低于10%,达到了农业机械作业标准所规定的旋耕作业农艺要求. 相似文献
9.
This paper addresses a nonlinear feedback control problem for the chaotic arch microelectro- mechanical system with unknown parameters, immeasurable states and partial state-constraint subjected to the distributed electrostatic actuation. To reflect inherent properties and design controller, the phase diagrams, bifurcation diagram and Poincare section are presented to investigate the nonlinear dynamics. The authors employ a symmetric barrier Lyapunov function to prevent violation of constraint when the arch micro-electro-mechanical system faces some limits. An RBF neural network system integrating with an update law is adopted to estimate unknown function with arbitrarily small error. To eliminate chaotic oscillation, a neuro-adaptive backstepping control scheme fused with an extended state tracking differentiator and an observer is constructed to lower requirements on measured states and precise system model. Besides, introducing an extended state tracking differentiator avoids repeated derivative for the virtual control signal associated with conventional backstepping. Finally, simulation results are presented to illustrate feasibility of the proposed scheme. 相似文献
10.
Lei Chen Cong-Fa Huang Yi-Cun Li Wei-Wei Deng Liang Mao Lei Wu Wen-Feng Zhang Lu Zhang Zhi-Jun Sun 《Cellular and molecular life sciences : CMLS》2018,75(11):2045-2058
The NLRP3 inflammasome is a critical innate immune pathway responsible for producing active interleukin (IL)-1β, which is associated with tumor development and immunity. However, the mechanisms regulating the inflammatory microenvironment, tumorigenesis and tumor immunity are unclear. Herein, we show that the NLRP3 inflammasome was over-expressed in human HNSCC tissues and that the IL-1β concentration was increased in the peripheral blood of HNSCC patients. Additionally, elevated NLRP3 inflammasome levels were detected in tumor tissues of Tgfbr1/Pten 2cKO HNSCC mice, and elevated IL-1β levels were detected in the peripheral blood serum, spleen, draining lymph nodes and tumor tissues. Blocking NLRP3 inflammasome activation using MCC950 remarkably reduced IL-1β production in an HNSCC mouse model and reduced the numbers of myeloid-derived suppressor cells (MDSCs), regulatory T cells (Tregs) and tumor-associated macrophages (TAMs). Moreover, inhibiting NLRP3 inflammasome activation increased the numbers of CD4+ and CD8+ T cells in HNSCC mice. Notably, the numbers of exhausted PD-1+ and Tim3+ T cells were significantly reduced. A human HNSCC tissue microarray showed that NLRP3 inflammasome expression was correlated with the expression of CD8 and CD4, the Treg marker Foxp3, the MDSC markers CD11b and CD33, and the TAM markers CD68 and CD163, PD-1 and Tim3. Overall, our results demonstrate that the NLRP3 inflammasome/IL-1β pathway promotes tumorigenesis in HNSCC and inactivation of this pathway delays tumor growth, accompanied by decreased immunosuppressive cell accumulation and an increased number of effector T cells. Thus, inhibition of the tumor microenvironment through the NLRP3 inflammasome/IL-1β pathway may provide a novel approach for HNSCC therapy. 相似文献